MCP Polyclonal Antibody, 100ul Endonucleases & Exonucleases Defects in this gene are

Defects in this gene are a cause of Niemann-Pick disease type A (NPA) and Niemann-Pick disease type B (NPB)

and cataract)

The beta and gamma actins co-exist in most cell types as components of the cytoskeleton| and as mediators of internal cell motility

This protein forms a trimeric complex with cyclin H and MAT1

subcellular location:Associated with perinuclear vesicles

MCP Polyclonal Antibody, 100ul Endonucleases & Exonucleases Defects in this gene areThe protein encoded by this gene is a type I membrane protein and is a regulatory part of the complement system. The encoded protein has cofactor activity for inactivation of complement components C3b and C4b by serum factor I, which protects the host cell from damage by complement. In addition, the encoded protein can act as a receptor for the Edmonston strain of measles virus, human herpesvirus 6, and type IV pili of pathogenic Neisseria. Finally,