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18 Tumor xenografts in NOD/SCID IL-2 receptor g-chain-null mice Xenograft tumors were generated by randomly injecting 110 6 MOLM14 shCTRL or shSLC7A11 cells into the tail veins of NOD/SCID IL-2 receptor g-chain-null mice (NSG) aged 69 weeks

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In the murine model of lipopolysaccharide (LPS)induced ALI, severely impaired FAO in alveolar EpiCs participated in the inflammatory response and lung injury, which might be attributed to the decreased expression of key mediators involved in FAO, such as CPT1A, and could partly be counteracted by treatment with a PPAR agonist, fenofibrate ( CPT1A aggravated EC apoptosis and lung injury, while treatment with L-carnitine attenuated the pathological changes (Yao et al., 2019)

The funders are not involved in the review design, reference collection, and decision to publish the manuscript

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